Enrichment of HP1a on Drosophila chromosome 4 genes creates an alternate chromatin structure critical for regulation in this heterochromatic domain.
Chromatin proteins captured by ChIP-mass spectrometry are linked to dosage compensation in Drosophila.
Comment on "Drosophila dosage compensation involves enhanced Pol II recruitment to male X-linked promoters".
Genome-wide chromatin interactions of the Nanog locus in pluripotency, differentiation, and reprogramming.
Variable requirements for DNA-binding proteins at polycomb-dependent repressive regions in human HOX clusters.
Epstein-Barr virus nuclear antigen leader protein localizes to promoters and enhancers with cell transcription factors and EBNA2.
"Jump start and gain" model for dosage compensation in Drosophila based on direct sequencing of nascent transcripts.
Epstein-Barr virus nuclear antigen 3C binds to BATF/IRF4 or SPI1/IRF4 composite sites and recruits Sin3A to repress CDKN2A.
Reciprocal interactions of human C10orf12 and C17orf96 with PRC2 revealed by BioTAP-XL cross-linking and affinity purification.
